Health

Hip dysplasia, Collie eye anomaly (CEA), and epilepsy are considered the primary genetic diseases of concern in the breed at this time. Collie eye anomaly (CEA) is a congenital, inherited eye disease affecting Border Collies and other breeds involving the retina, choroid, and sclera. In Border Collies, it is generally a mild disease and rarely significantly impairs vision. There is now a DNA test available for CEA and, through its use, breeders can ensure that they will not produce affected pups. There are no genetic tests available for hip dysplasia or epilepsy, although careful breeding practices are known to lower the incidence of both.

Elbow dysplasia or Osteochondritis, deafness, and hypothyroidism may also occur in the breed. Dogs homozygous for the merle gene are likely to have eye and/or hearing problems. Responsible breeders do not mate merles to one another.

Neuronal ceroid lipofuscinosis (NCL) is a rare but serious disease. NCL results in severe neurological impairment and early death; afflicted dogs rarely survive beyond two years of age. The mutation causing the form of the disease found in Border Collies was identified by Scott Melville in the laboratory of Dr. Alan Wilton of the School of Biotechnology and Biomolecular Sciences, University of New South Wales, Sydney, Australia. There is no treatment or cure, but a DNA test is now available to detect carriers as well as affected dogs.

Trapped Neutrophil Syndrome (TNS) is a hereditary disease which inhibits the release of neutrophils produced in the bone marrow into the blood stream. Puppies affected with this disease will eventually succumb to infection. Because this is an autoimmune-deficiency disease the puppies present a variety of symptoms depending upon what infections they fall susceptible to, and so it has gone undiagnosed in the past. Once thought to be rare, it is now believed to be responsible for many cases of "fading puppies". There is no cure, but a DNA test is now available to detect carriers as well as affected dogs. Please visit the American Border Collie Association for more information.

Health

Dalmatians are relatively a healthy and easy to keep breed. Like other breeds, Dalmatians display a propensity towards certain health problems specific to their breed, such as deafness, allergies and urinary stones. Reputable breeders have their puppies BAER (Brainstem Auditory Evoked Response) tested to ensure the status of the hearing on their pups. Hip dysplasia is not a major issue in this breed. In their late teens, both males and females may suffer bone spurs and arthritic conditions. Autoimmune thyroiditis may be a relatively common condition for the breed.

A genetic predisposition for deafness is a serious health problem for Dalmatians; American Dalmatians exhibit a prevalence for bilateral congenital sensoneural deafness of 8% (for which there is no possible treatment), compared with 5.3% for the UK population. Deafness was not recognized by early breeders, so the breed was thought to be unintelligent. Many breeders, when hearing testing started to become the norm, were amazed to discover that they had unilateral hearing dogs. Even after recognizing the problem as a genetic fault, breeders did not understand the dogs' nature, and deafness in Dalmatians continues to be a frequent problem.

Dalmatians, like humans, can suffer from hyperuricemia. Dalmatians' livers have trouble breaking down uric acid, which can build up in the blood serum (hyperuricemia) causing gout. Uric acid can also be excreted in high concentration into the urine, causing kidney stones and bladder stones. These conditions are most likely to occur in middle-aged males. Males over ten are prone to kidney stones and should have their calcium intake reduced or be given preventive medication. To reduce the risk of gout and stones, owners should carefully limit the intake of purines by avoiding giving their dogs food containing organ meats, animal byproducts, or other high-purine ingredients. Hyperuricemia in Dalmatians responds to treatment with orgotein, the veterinary formulation of the antioxidant enzyme superoxide dismutase.